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The study also revealed the underlying mechanism: autoantibodies and IFN- in SLE serum enhance the binding of the transcription suppressor CREM to the Gpx4 promoter, which in turn suppresses Gpx4 expression

173 Therefore, it has therapeutic potential in the treatment of CNS diseases such as neurodegenerative diseases, epilepsy, anxiety, and depression, without simultaneous psychotic adverse effects

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doi: 10.2147/DDDT.S168878 82 ZhaoMChenJ-YChuY-DZhuY-BLuoLBuS-Z

By regulating the Nrf2/HO-1 pathway, A5 improves the activity of SOD and GPX and the level of glutathione, reduces the levels of iron deposition, 4-HNE and MDA, thereby improving the ferroptosis of neurons induced by TBI

Systemic oxidative stress in non-arteritic anterior ischemic optic neuropathy