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The literature includes its role in wound healing, angiogenesis, and contexts related to fibrosis and remodeling [2][5]

This change is one of the mechanisms underlying long-term potentiation (LTP)

demonstrated that targeting NADPH oxidase-mediated ROS release and ferroptosis accumulation can improve septic kidney injury in LPS-stimulated HFD-induced diabetic mice and may also be a potential therapeutic target [138]

Relationships of generalized and regional adiposity to insulin sensitivity in men

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