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Cavestro, C

The problem is, it's an access issue

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This is the piece most standalone BPC-157 protocols miss: better-organised connective tissue means lower re-injury risk long after the acute repair phase ends

The role of UCP2 in controlling GSIS was thought to dissipate proton conductance and hence inhibit a rise in cytosolic ATP, blunting insulin secretion [85]

PMID 25875118
