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Deficiency in mitochondrial complex I can result in oxidative stress, heightening neuronal susceptibility to excitotoxic injury

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doi: 10.1021/ar200260p

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This position is supported by in vivo studies, which used genetically encoded redox sensors to investigate the spatial patterning of cellular redox potential in Caenorhabditis elegans [11]

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And so that, uh, that stack

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[DOI] [PubMed] [Google Scholar] [181].Gilbert HF, Molecular and cellular aspects of thiol-disulfide exchange, Adv

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4 : 693696 Takagi Y, Mitsui A, Nishiyama A, Nozaki K, Sono H, Gon Y, Hashimoto N and Yodoi J (1999) Overexpression of thioredoxin in transgenic mice attenuates focal ischemic brain damage

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