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Antimicrobial peptides and the gut microbiome in inflammatory bowel disease

Inhibiting the elevated expression of GJ components in hepsin / mice reduces APAP toxicity by limiting oxidative stress in the liver To investigate GJ expression variations after APAP administration in wild-type and hepsin / mice, immunofluorescence staining for Cx32 was assessed at different time points

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These findings elucidate a novel MFAP2-ITGB8-FAK-ERK1/2-ETS2-CYP27A1-LXR signaling axis, significantly activated by CAFs-derived MFAP2 in both in vitro and in vivo models, contributing to immune exhaustion and tumor progression