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Potential compensatory mechanisms correcting underlying vulnerability may be speculated as one of the reasons for such discrepancies, consistently with previous findings showing decrease in C8 and C10 after antidepressant treatment [49]

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Since people with cocaine use disorder suffer from depression, such comorbidity was evaluated using cocaine self-administration/extinction/reinstatement procedures in the rat model of depression based on the removal of the olfactory bulbs [19]

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