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The nuclear form of ATF6 inhibits the transcriptional activity of sterol regulatory element-binding protein 2 (SREBP2) and blocking ATF6 inhibited hepatic mitochondrial fatty acid oxidation by decreasing the transcriptional activity of PPAR [62]

GPCRs exhibit distinctive combinations of signal-transduction capabilities involving G protein-dependent signaling pathways, as well as G protein-independent signaling pathways, and complex regulation processes, despite the fact that their structural similarities

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