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8 Therapeutic perspective targeting propionate metabolism in PA Since chronically elevated propionate is a key factor in the neurological dysfunction associated with PA, treatment strategies should focus on reducing propionate synthesis and enhancing its metabolic disposal

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Genotype 1 is by far the most frequent genotype in chronically infected patients worldwide, with subtypes 1a and 1b representing the vast majority of circulating strains [2], [3], [4]

Hydrogen Sulfide Ame-liorated High Choline-Induced Cardiac Dysfunction by Inhibiting cGAS-STING-NLRP3 Inflammasome Pathway

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