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Hyperphosphorylation of the Rabs disrupts their interaction with GTP/GDP exchange factors (GEFs), and Rab GDP dissociation inhibitors (GDIs), resulting in their inactivation and membrane-cytosol redistribution (Steger et al., 2016)

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The possible complementary role in the etiology of 1 of this patient's medications could explain these findings, and the histological changes associated with the drug reaction may have masked those more often found in cases of vitamin B12 deficiency

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