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Pediatr Res 70(1):6166 Oliveira MS, Floriano EM, Mazin SC, Martinez EZ, de Vicente WV, A, Peres LC, Rossi MA, Ramos SG (2011) Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms

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Kuramochi cells, which are sensitive to DNA-damaging agents but modestly HR proficient 27 , also showed increased RAD51 foci following treatment with DNA-damaging agents combined with KG or carnitine supplementation (Extended Data Fig

UA suppresses carnitine palmitoyl transferase 1B (CPT1B), thereby inhibiting FA transport into the mitochondrial inner matrix for elimination