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In summary, mitochondrial OCM primarily uses serine as a one-carbon source and plays a critical role in supporting biosynthetic processes such as nucleotide synthesis, protein synthesis and methylation, particularly under conditions of high proliferative demand and cellular stress [17]

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Disrupted iron balance elevates ferrous ion concentrations, driving hydroxyl radical generation through Fenton chemistry that subsequently induces widespread phospholipid peroxidation within neural cells (152)

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