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Triptolide induces apoptosis and cytoprotective autophagy by ROS accumulation via directly targeting peroxiredoxin 2 in gastric cancer cells

Moreover, excessive reactive oxygen species (ROS) and inflammatory cytokines secreted from macrophages and other immune cells [e.g., interferon- (IFN) and tumor necrosis factor (TNF)] can arrest the anagen phase, driving premature telogen and catagen phases, inducing apoptosis in the follicular microenvironment, leading to miniaturization and reduced hair density [2, 4, 5]

175 These findings support further use of PROM2-targeted intervention strategies in treating metastatic melanoma and other cancers

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Pharmacological actions of statins: a critical appraisal in the management of cancer