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There was a report suggested that the elevated ROS causing OS are produced by erythrocytes and apoptotic endometrioma cells, as well as the activated macrophages that are recruited to phagocytize apoptotic cells [10]

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AML cells are completely dependent on exogenous GLN, and knockdown of high-affinity ASCT2 leads to apoptosis in AML cell lines and inhibits tumor progression in AML xenografts and primary AML mouse models [47] Indeed, ASCT2 plays pleiotropic roles in cellular metabolism and serves as a promising molecular target for the treatment of leukemia [27]

[PubMed] [Google Scholar] 42.Savica V., Santoro D., Mazzaglia G., Ciolino F., Monardo P., Calvani M., Bellinghieri G., Kopple J.D

Mechanism of oxidative stress p38MAPK-SGK1 signaling axis in experimental autoimmune encephalomyelitis (EAE)

Homocysteine increases the risk of mortality in elderly individuals