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Collectively, these mitochondrial dysfunction-associated events contribute to ferroptosis-related nucleus pulposus cell injury and extracellular matrix degradation in IDD, thereby suggesting that targeting mitochondrial dysfunction may represent a potential therapeutic strategy for IDD

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ACTH-receptor expression, regulation and role in adrenocortial tumor formation

Certain organs that are rich in iron and lipids exhibit increased susceptibility to ferroptosis induction during the process of carcinogenesis, particularly in the context of glioblastoma, pancreatic cancer, and triple-negative breast cancer [8385]