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Mechanistically, SAC directly interacts with Keap1 to promote Nrf2 into the nucleus, enhance the transcription of antioxidant genes (such as GPX4 and NQO1), and restore the biosynthesis of GSH

Hedrick et al., 2006)

Kmail et al., 2025)

Moreover, the method is robust, since it can be adapted to different feeder models, and the model can be trained via a synthetic data set and still achieves desirable performance in real-world measurement

The main cytotoxic action of TMZ is due to the formation of 6-methylguanine lesions which causes apoptosis, autophagy, and cellular senescence of tumor cells [32]

A., Kohli, S., Bondy, G