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Patients with TCMR demonstrate higher miR-223, miR-10a (72), miR-99a, and miR-100 levels in blood samples (73), but lower levels of miR-99a expression in kidney allografts (74, 75), implying a possible difference in miR levels between renal tissue and blood samples

These bioenergetic deficits contribute to muscle and neuronal fatigue ( This scoping review aims to examine the role of oxidative stress and mitochondrial dysfunction in FM, with a focus on redox imbalance, neuroinflammation, and potential therapeutic interventions targeting these pathways

However, the complex machinery underlying CIPN is poorly understood

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