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Our data revealed that HuR has the most decisive impact on the KGA/GAC protein level on a cell background that is glutamine withdraw and glutaminase-inhibition resistant (which even includes some TN tumor cells), implying that it is not the HuR levels by itself, but glutamine-dependent TCA cycle itself is the critical factor behind HuR having the most pronounced effect over KGA/GAC levels

R., Stringer K

Cancer Cell 32 , 807823.12 (2017)

21 This toxin-mediated glucosylation locks the Rho family GTPases in an inactive conformation, which then affects a number of important downstream cellular pathways

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