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A commonly agreed upon the fact in the pathogenesis of SLE is the dysfunction of T cells, B cells, dendritic cells, macrophages, and neutrophils, likely triggered by genes and/or the environment [4]

doi: 10.1016/j.thromres.2020.05.031

BMC Ophthalmol 20:73

However, l-serine supplementation does not reduce hyperglycemia in BKS- db/db models despite the improvement in neuropathy phenotypes, suggesting that some forms of diabetic peripheral neuropathy are more directly driven by l-serine deficiency as opposed to hyperglycemia

Lazo M, Clark JM

TRIM25 regulates oxaliplatin resistance in colorectal cancer by promoting EZH2 stability