glutathione and metabolism in Brain Disorders Aging 8 An outline of the

TO: contributed to the study design, writing, and editing

Int Immunopharmacol 99:108002

As oxidative stress increases, further stimulation of pro-inflammatory cytokines such as TNF- and IL-1 increases and contributes to synaptic dysfunction and neuronal degeneration ( These studies suggest that the loss of antioxidant defense is an early biochemical event rather than a late-stage consequence of neuronal death

Results Establishment of anti-VEGF therapyresistant model by overexpressing VEGFR2-Fc To establish anti-VEGF therapyresistant models, we introduced Mock vector or an expression vector for the VEGFR2-Fc fusion gene (Supplementary Fig

Fortunato F, Martinelli D, Lo Caputo S, Santantonio T, Dattoli V, Lopalco PL, et al

Curr Ther Res Clin Exp 66: 13-22