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Depletion likely results from the intracellular utilization of GSH as a result of EtOH exposure, as well as, in the liver, efflux of GSH from hepatocytes into the blood

Prolonged aluminum exposure increases A levels and oxidative damage

doi: 10.1016/j.jss.2012.06.024

J Endocrinol (2014) 221:R3140

10.1007/BF02537398

Baquero, F
