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78 It was found that reduced GPX4 expression leads to increased lipid ROS levels and activation of ferroptosis

Comprehensive evaluation of lipopolysaccharide-induced changes in rats based on metabolomics

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This hypothesis posits that with steatosis, the liver undergoes ER stress, oxidative stress and mitochondrial dysfunction, leading to an increase in oxidative metabolites, hepatocellular damage and secretion of a large number of inflammatory factors, which ultimately leads to further deterioration into liver disease, liver fibrosis, cirrhosis, inflammatory necrosis and other reactions (19,20)

Thus, the Nrf2 pathway may be a potential option for treating liver diseases

The oxidation steps, utilizing glucose-6-phosphate (G6P) as the substrate, occur at the beginning of the pathway and are the reactions that generate NADPH