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doi: 10.1017/S0029665118001192 250
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543 Our previous research has shown that programmed exercise can inhibit pathological ventricular hypertrophy and myocardial fibrosis gene expression through the suppression of PKC-/NFAT signaling in a mouse model
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Exploratory analysis of biochemical secondary endpoints Exploratory data (GSH, sulfur and amino acid compounds) are presented as medians with interquartile range [Q1-Q3]
