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This process plays a central role in the progression of pulmonary fibrosis, highlighting the regulatory interplay between lipid metabolism and ferroptosis as a potential therapeutic target for future anti-fibrotic strategies

[DOI] [PMC free article] [PubMed] [Google Scholar] 106.Walker HK, Hall WD and Hurst JW, Clinical Methods: The History, Physical, and Laboratory Examinations, Butterworths, Boston, 1990, p

Histone deacetylase inhibitor restores surfactant protein-C expression in alveolar-epithelial type II cells and attenuates bleomycin-induced pulmonary fibrosis in vivo

Retrieved 28 January 2014

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