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The brain pathology of this disease is similar to that seen in oxidative damage resulting from sulfhydryl depletion due to chronic mercury poisoning (Minamata disease) and is also compatible with decreased cerebral glutathione levels (09)

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Our mechanistic investigations revealed that intracellular GSSG elevation sensitizes hepatocytes to TNF killing by inducing IKK--SSG formation, leading to suppressed NF-B activation

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And the peptides ability to attract stem cells and stimulate fibroblast proliferation provides the cellular workforce needed for tissue reconstruction

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When it is exposed to heat and light, its antioxidant activity is reduced

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