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oxalates and glutathione Gut microbiota-bile acid crosstalk contributes to calcium oxalate nephropathy through Hsp90α-mediated ferroptosis PRMT1-mediated methylation of UBE2m promoting

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10.1016/j.biopha.2020.110735 33 HeldtH.-W.PiechullaB

oxalates and glutathione Gut microbiota-bile acid crosstalk contributes to calcium oxalate nephropathy through Hsp90-mediated ferroptosis PRMT1-mediated methylation of UBE2m promoting

This phase 3 trial directly compares CagriSema 2.4 mg/2.4 mg against tirzepatide 15 mg in approximately 800 adults with obesity

oxalates and glutathione Gut microbiota-bile acid crosstalk contributes to calcium oxalate nephropathy through Hsp90-mediated ferroptosis PRMT1-mediated methylation of UBE2m promoting

ABCC5 increased intracellular GSH and attenuated lipid peroxidation accumulation by stabilizing SLC7A11 protein, which inhibited ferroptosis (Huang et al., 2021)

oxalates and glutathione Gut microbiota-bile acid crosstalk contributes to calcium oxalate nephropathy through Hsp90-mediated ferroptosis PRMT1-mediated methylation of UBE2m promoting

Authors' contributions HN, MR, JJ, and CM formulated the questions and contributed substantially to the design of the project

oxalates and glutathione Gut microbiota-bile acid crosstalk contributes to calcium oxalate nephropathy through Hsp90-mediated ferroptosis PRMT1-mediated methylation of UBE2m promoting

Yalshetti et al

oxalates and glutathione Gut microbiota-bile acid crosstalk contributes to calcium oxalate nephropathy through Hsp90-mediated ferroptosis PRMT1-mediated methylation of UBE2m promoting

10.1016/j.biomaterials.2019.119357 57 DeeksS

oxalates and glutathione Gut microbiota-bile acid crosstalk contributes to calcium oxalate nephropathy through Hsp90-mediated ferroptosis PRMT1-mediated methylation of UBE2m promoting

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