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5a), supporting the case that LCS3 exerts toxicity by disrupting both redox pathways

Mechanistically, GAS5 inhibits proliferation and promotes apoptosis through the regulation of E2F1, p21, and p53, and suppresses oncogenic pathways including EGFR, MAPK, and AKT

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The first change seen is endothelial activation, which is characterized by an aberrant pro-inflammatory and pro-thrombotic phenotype of blood vessel endothelial cells