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The schematic diagram of radiationinduced cell cycle arrest and DNA damage repair is shown in Figure 1

Perhaps the most profound biological driver of chronic Lyme symptoms is post-infectious autoimmunity and neuroinflammation

3.6.2 GILZ The signal of mutual inhibition between MAPK and GR also involves the expression of anti-inflammatory genes such as GILZ

26 showed that the core subunit NDUFS1 of complex I was oxidatively damaged in PD brains, resulting in complex I misassembling and functional impairment

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It also modulates neurotransmitters like dopamine and serotonin, which are essential for mood, motivation, and executive function
