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Its core mechanism is actin sequestration: it binds G-actin monomers, blocking polymerisation, which is exactly the condition cells need to migrate, contract wounds, and build new vessels

[DOI] [Google Scholar] 80.Umemura M., Ueda H., Tomono K., Nagai T

For gastrointestinal applications, oral administration becomes viable due to BPC-157s unusual stability in gastric juice

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Can BPC-157 replace medical treatment
