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We have found that Sema4D-induced nuclear translocation of AR is dependent on Ran signalling and the GAP domain of PlexinB1, suggesting that PlexinB1/B2 contribute to ADT resistance through their function as RanGAPs

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It is studied for how it speaks to tissue-repair signalling and angiogenesis

doi:10.3390/ijms19071987 12

Oxid Med Cell Longev (2019) 2019:9151067
