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In ethanol sensitized hepatocytes, TNF- increased the susceptibility of hepatocytes after mitochondrial GSH depletion
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Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer's disease models and human brains
MLCK-dependent occludin endocytosis via a caveolar process promotes leak pathway permeability [19]
