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This resistance allows cancer cells to tolerate ROS accumulation and maintain survival unless the balance is further disrupted by external ROS inducers (e.g., H 2 O 2 ) or inhibitors of ferroptosis-resistance mechanisms (e.g., RSL3 and Erastin)

The frameshift mutations in P1, P2, P3, P4, and P5 all produce truncated transcripts that are predicted to be prone to nonsense-mediated mRNA decay (NMD)

Int J Mol Sci 20:131

Confirmation of this hypothesis of juglone-glutathione conjugates being the inhibitory molecules will thus have to await the availability of these conjugates