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Lansdown, A
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10.1016/S0092-8674(02)00613-X [DOI] [PubMed] [Google Scholar] He L., Kim J
siRNA-mediated knockdown of BACH1 reversed the increased proliferation of HUVECs after BPC157 treatment (Fig
The present 7 T MRS study has two primary objectives: (1) to compare brain metabolite levels in ASD with controls, and (2) to investigate the association between neurotransmission disturbances and ASD symptomatology
Untreated cells in 2% or 10% FBS DMEM for 3 days were used as negative controls
