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Cancer-derived p53 mutants suppress p53-target gene expressionpotential mechanism for gain of function of mutant p53

doi: 10.1038/s41380-022-01741-1

Aberrant polyadenylation events within introns of tumor suppressor genes such as DICER1 and FOXN3 result in the production of truncated proteins lacking essential tumor-suppressive functions, thereby promoting tumorigenesis

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These data together with those obtained for 2-VP [14,15] let us to select NEM as the most suitable alkylating agent to protect the oxidation of thiol groups, according to what suggested previously [Butler, Tietze]