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thus, after exceeding this concentration of edelfosine, excess NBD-Sph can participate in micelle formation, which is most likely what is observed as an intensity increase after the addition of 1 M edelfosine to 1 M NBD-Sph (Fig S2C)
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Similar to the mechanism underlying coronary calcification, lipid deposition, inflammatory cell infiltration, extracellular matrix dysregulation, and the osteogenic transformation of valvular interstitial cells contribute to the pathophysiology of this disease [74]

Mitochondrial dysfunction and the pathophysiology of myalgic encephalomyelitis/chronic fatigue syndrome (Me/Cfs)
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