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Further investigations revealed that ATF2 overexpression significantly elevated both CENPT mRNA and protein levels (fig S11E/G), while ATF2 knockdown (sh-ATF2) suppressed CENPT expression in RCC cell lines (fig S11F/H)

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This kind of therapy would exert its beneficial action through antioxidant and immunoregulatory functions, optimizing mitochondrial function and even increasing the biogenesis of this vital organelle

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