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C.MacanderL.JarkowskiA.et al

Hum Genet 69:88

The released GHK-Cu promoted bone marrow stem cell proliferation, induced neovascularization, and facilitated macrophage polarization toward the M2 (anti-inflammatory, pro-repair) phenotype

Together with the designed sequences (E1, ES2, ES2r1 and ES2r2), a triple repeat of HIV-TAT, labelled as ES1, was recruited to the same in silico workflow, producing a comparable CR3 affinity with E1 (Table 1)

Redox control of protein degradation

Divide doses for improved tolerance
