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The potential utility of incorporating this mechanism into complex LCR/beta-globin LVs was suggested by our discovery that unwanted RNA splicing is the key determinant of LCR/beta-globin -RV instability and low titers, and that this problem could be largely corrected by the removal of undesirable splice sites [131]

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In the kidneys, the peptide directly interferes with a major cause of the problem which is p38, Lucas says, noting that p38 is known to play an important role in cells that line blood vessels and the heart, and like those lining the glomeruli

10.1016/j.cell.2004.05.008 Cell 141 RochaE

Loss of endothelial glycocalyx during acute hyperglycemia coincides with endothelial dysfunction and coagulation activation in vivo