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Therefore, CIPN is regarded a multifactorial disorder with diverse pharmacological mechanisms, and a number of common pathologies, including altered ion channel functions, oxidative stress, axonal degeneration, impaired calcium homeostasis, neuroinflammation, and activation of the immune system, have been proposed to underlie CIPN development (Fig

PennellDJPorterJBCappelliniMDChanLLEl-BeshlawyAAydinokYet alDeferasirox for up to 3 years leads to continued improvement of myocardial T2* in patients with -thalassemia major

Glutathione S-Transferase Pi 1 (GSTP1) Gene 313 A/G (rs1695) polymorphism is associated with the risk of urinary bladder cancer: Evidence from a systematic review and meta-analysis based on 34 case-control studies

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Downregulation was observed in 22% of the studies with susceptible plants and in 17% of the studies with resistant, partially resistant or tolerant plants (Yang et al., 2006