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The findings of the current study suggest that CIT in cancer patients results from HSCs losing their capacity for self-renewal, HPCs apoptosis, and BMECs dysfunction (Fig

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It provides evidence that CtAtm1, a homolog of human ABCB7, facilitates the delivery from glutathione-complexed iron-sulfur clusters, enhancing our comprehension of the transport mechanisms of eukaryotic ABCB7-type proteins

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