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High hyperdiploidy generally occurs in cases with clinically favorable prognostic factors (patients aged 1 to 80 chromosomes) are much less common and appear to be biologically distinct from high hyperdiploidy.[33] Unlike high hyperdiploidy, a high proportion of near tetraploid cases harbor a cryptic ETV6 :: RUNX1 fusion.[33-35] Near triploidy and tetraploidy were previously thought to be associated with an unfavorable prognosis, but later studies suggest that this may not be the case.[33,35] The genomic landscape of hyperdiploid ALL is characterized by variants in genes of the receptor tyrosine kinase (RTK)/RAS pathway in approximately one-half of cases

in addition to measuring collagen, collagen fragments, and -SMA as distinct biomarkers for establishing fibrosis

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263272.Google Scholar Sun, QY (2003) Cellular and molecular mechanisms leading to cortical reaction and polyspermy block in mammalian eggs

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