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Description
Theres ongoing research in this area

Elevated reactive oxygen species and activated FOXO3a in Rictor -deleted cells strikingly shifts the functional interaction of -catenin from TCF to FOXO3a, which strongly inhibits classical Wnt/-catenin signaling
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and pharmacol

Even though the definitive role of GSH imbalance in PD is still debated, the efficacy of therapeutic antioxidant and anti-inflammatory agents in PD models underscores the potential of targeting this pathway to halt disease progression (Leathem et al., 2022)

It is a subject of ongoing research for its potential regenerative properties in vitro