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(2003) published findings in Bulletin of Experimental Biology and Medicine examining Epithalons role in telomerase activation in human somatic cells one of the most cited early investigations into this peptides mechanism

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Additionally, obesity and IR both contribute to -cell dysfunction and apoptosis, mechanistically explained by glucotoxicity and increased immunogenicity (the accelerator hypothesis) or by -cell exhaustion due to increased metabolic demand (the overload hypothesis) [23]

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