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These findings lead us to speculate that, during the course of infection, and on the assumption that intermediate-sized particles of chitin have been produced during the first stages of the infection, inflammation and destruction of the fungus are induced to promote the elimination of the invading organism, but once the fungus has been killed, the chitin particles found will mostly be small, which will then trigger an anti-inflammatory response through the production of IL-10, mitigating inflammation and therefore avoiding excessive tissue injury [36]

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Nevertheless, as the unnecessary side effects of intravenous NGF on the peripheral nerve are limited by its low stability, short half-life, and high cost (89, 90), it is not encouraged to try exogenous NGF in humans (91)