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Description
These mutations exacerbate the production of ROS by disrupting the assembly of respiratory chain complexes and impairing the function of ATP synthase, subsequently activating apoptotic signaling pathways
Their activation patterns across disorders illustrate both harmful and protective influences
201 The results indicated that the modified system loaded with the drug exhibited a higher cytotoxic effect compared with the free drug
Metabolic reprogramming of macrophages during infections and cancer
Mechanisms of mitochondrial respiratory adaptation
The activation of the RAS-RAF-MAPK pathway produces eicosanoids (including leukotrienes C4 and prostaglandin D2) as well as cytokines
