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Concurrently, stress-activated signaling nodes such as GATA2/NF-B/STAT3 and Yes-associated proteins (YAP)/TEA domain (TEAD) reinforce survival through cross-talk with epigenetic modifiers, stabilizing DTP phenotypes [39, 41, 42] (Table 3)

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Later on, this was proved in the OVX mouse model that showed GM dysbiosis due to estrogen deficiency brought about by integral bone loss through enhanced gut permeability alongside excessive expression of cytokines, which orchestrated osteoclastogenesis

Ventzel L, Jensen AB, Jensen AR, Jensen TS, Finnerup NB

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Mitochondrial Disorder: Kearns-Sayre Syndrome