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Funding This work was supported by the National Natural Science Foundation of China (81570059, 31370524) and the Natural Science Foundation of Jiangsu Province of China (BK20151398)

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Met activation has been extensively shown to drive proliferation, migration, invasion, and angiogenesis in HNSCC and other tumor types (16) and HGF/Met activation is a known mechanism of resistance to anti-EGFR therapy (17)