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Under hypoxic conditions, the O 2 -dependent PHD is inhibited leading to stabilization and translocation of HIF into the nucleus [17]

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The reaction could not be explained by other variables such as location, season, or other medications taken at the same time

[ 197 ] Another study suggests that the inhibition of BACH1 attenuates oxidative stressinduced osteoblast apoptosis and impairment via the amplification of NRF2/ARE signaling, highlighting the BACH1/NRF2/ARE regulatory axis in the development of osteoporosis