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doi: 10.2337/diacare.15.1.1

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Its mechanism may related to Ng-Ft1 selectively targeted the deubitinating enzyme ubiquitin-specific protease 9X (USP9X), disrupting its protective effect on -catenin, leading to reduce the expression of downstream genes of the Wnt/-catenin signaling pathway

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The specific challenge that was resolved, was the activation of the collateral pathways and upgrading of the particular minor vessel (i.e., remaining episcleral vein that was not cauterized) [11]