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Confirming the significant role of Met35 of A1742 in these observations, when the Met35 residue was substituted by norleucine (as noted above, CH 2 in place of the S-atom of Met), no oxidative damage or neurotoxicity to hippocampal neurons was found (115)

Krishnamurthy, J

Consistent with this, blocking either c-Maf or MafG induction during BDL protected against the fall in expression of GSH synthetic enzymes, GSH levels and BDL-induced liver injury [90]