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An initial support was provided by showing that MAO-A expression is increased in the rat heart in an age-dependent manner [32]

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Eventually, a primary dysfunction of NMDARs, or neuroinflammation, or mitochondrial dysfunction, or impaired regulation of antioxidant systems could involve in turn the other vicious circles, all converging to common deleterious impacts on PV neurons and oligodendrocytes during neurodevelopment (Fig